CI

At a glance

ClinicalIndex Comparison Record
N/ACompleted· 80 enrolled
Drug / intervention
Physostigminedrug
Likely dose
Not stated in record
Structured eligibility isn't available for this trial yet — see the full criteria in the Eligibility tab below.

Standardized by ClinicalIndex from the ClinicalTrials.gov record · verify against the source.

Search/NCT02008292
NCT02008292N/ACompleted

Acetylcholine, Tobacco Smoking, Genes and Nicotinic Receptors

Yale University·interventional·Posted Dec 11, 2013·Updated Oct 25, 2023

In Brief

A clinical study evaluating Physostigmine for Smoking and Schizophrenia. Completed, enrolled 80 participants across 3 sites.

Detailed Summary

The purpose of the study is to measure the sensitivity of NCFHEB binding to changes in endogenous acetylcholine levels in healthy smoking and nonsmoking subjects, and in schizophrenic smoking and nonsmoking subjects. We hypothesize that physostigmine-induced elevated ACh levels will lead to a reduction in the availability of nicotinic receptors for the binding of the radioligand. We hypothesize there will be greater increase in ACh level (or greater reduction in radio tracer binding) in smoking as compared to nonsmoking subjects. We hypothesize there will be greater increase in ACh level (or greater reduction in radio tracer binding) in smoking as compared to nonsmoking subjects with schizophrenia, but the extent of this change will be different than in controls. We are also measuring the sensitivity of PHNO binding to changes in dopamine levels in healthy smoking and nonsmoking subjects before and after amphetamine challenge.

Study Details

Study Typeinterventional
Allocation--
Masking--
Primary Purpose--
CountriesUnited States

Timeline

N/ACompletedFinished
2014201520162017201820192020202120222023202420252026
First PostedDec 11, 2013
Enrollment StartSep 1, 2013
Primary CompletionDec 1, 2021
TodayJul 2, 2026
Enrollment to primary: 8.3 yearsPosted 12.6 years ago

Interventions

Physostigminedrug

All subjects will receive physostigmine to induce elevated ACh levels in the brain.