CI

At a glance

ClinicalIndex Comparison Record
Phase 2Active· 26 enrolled / 26 target
Drug / intervention
Zabadinostat (CXD101) and Geptanolimab +1 moredrug
Likely dose
Zabadinostat (CXD101) and Geptanolimab 20mgfrom record
Key inclusion· 3
  • Diagnosis of HCC according to AASLD guideline
  • Prior systemic treatment with immune checkpoint inhibitors (anti-PD1, anti-PDL1 or anti-CTLA4)
  • Previous ICI duration of 6 weeks or longer
Key exclusion· 11
  • Previous severe autoimmune complications from immune checkpoint inhibitors
  • History of moderate to severe autoimmune disease requiring steroid use
  • History of organ transplant
  • Prior use of lenvatinib or sorafenib

Standardized by ClinicalIndex from the ClinicalTrials.gov record · verify against the source.

Search/NCT05873244
NCT05873244Phase 2ActiveOn Track (0.8/mo)

Epigenetic Therapeutics to Overcome Resistance Against Immune Checkpoint Inhibitors in Hepatocellular Carcinoma: A Proof-of-concept Clinical Trial

Stephen Chan Lam·interventional·Posted May 24, 2023·Updated Jun 18, 2026

In Brief

A Phase 2 clinical trial evaluating Zabadinostat (CXD101) and Geptanolimab and Lenvatinib and Sorafenib for HCC. Active but no longer recruiting, targeting 26 participants across 2 sites.

Detailed Summary

For hepatocellular carcinoma (HCC), durable responses and improved survivals have been reported in clinical trials on immune checkpoint inhibitor (ICI)-based treatment. However, resistance to ICI is increasingly encountered in clinical practice in HCC patients. Various approaches are currently evaluated in clinical setting to tackle acquired resistance during treatment of ICIs in HCC. Our group has a track record of studying the role of histone deacetylases (HDACs) in mediating resistance to ICI in HCC. First, based on single-cell sequencing data of serial biopsy of tumor in our phase II clinical trial on pembrolizumab in HCC (NCT03419481), the investigators reveal an upregulation of class 1 HDAC in patients with acquired resistance to pembrolizumab, which was associated with reduced lymphoid/myeloid cellular ratio in the tumor. Further, the investigators showed that HDAC8, a class 1 HDAC, could diminish the efficacy of anti-programmed cell death (ligand)-1 (PD\[L\]-1) by the mechanism of T-cell exclusion from the tumor environment (SciTranl Med. 2021;13:online). Finally, the investigators combine CXD101, a potent selective class I HDAC inhibitor, with anti-PD(L)-1 in orthotopic immunocompetent HCC mouse model with resistance to anti-PD(L)-1 treatment and find that the combination regimen could reverse the resistance phenotype and significantly improve survivals of mice than either CXD101 or anti-PD(L)-1 alone.

Study Details

Study Typeinterventional
Allocation--
Masking--
Primary Purpose--
ConditionsHCC
CountriesHong Kong
Collaborators--

Timeline

Phase 2Active
202420252026202720282029
First PostedMay 24, 2023
Enrollment StartAug 21, 2023
Primary CompletionDec 30, 2027
Study CompletionDec 30, 2028
TodayJul 2, 2026
Enrollment to primary: 4.4 yearsPosted 3.1 years agoPrimary completion in 1.5 years

Arms & Interventions

Experiment armexperimental

* Zabadinostat (CXD101) at 20mg twice daily per orally Day 1-5 every 3 weeks * Geptanolimab at 3mg/kg given intravenously every 2 weeks

Drug: Zabadinostat (CXD101) and Geptanolimab
Control armother

Clinicians' choice of TKI at corresponding recommended dosage: * Lenvatinib at 8mg daily for patients with body weight \<60kg or 12mg daily with body weight ≥ 60kg * Sorafenib at 400mg twice daily

Drug: Lenvatinib and Sorafenib

Interventions

Zabadinostat (CXD101) and Geptanolimabdrug

* Zabadinostat (CXD101) at 20mg twice daily per orally Day 1-5 every 3 weeks * Geptanolimab at 3mg/kg given intravenously every 2 weeks

Lenvatinib and Sorafenibdrug

Clinicians' choice of TKI at corresponding recommended dosage: * Lenvatinib at 8mg daily for patients with body weight \<60kg or 12mg daily with body weight ≥ 60kg * Sorafenib at 400mg twice daily